The testing of anti-stress drugs, scientists have discovered a remedy for baldness


Investigating the impact of anti-stress drugs in the digestive tract, scientists from the University of California at Los Angeles unexpectedly discovered a remedy for baldness.

In the series A — mice before the experiment. In a series of in — they are just three days after a five-day series of injections. In line with — they are four weeks

In the experiments, they used genetically altered mice suffering from overproduction of the stress hormone CRF (corticotrophin-releasing factor). Such mice usually begin to age rapidly balding. The mice were injected with the drug astressin-B, which blocks the action of CRF. Three months later, they decided to hold a gastroenterological examination of mice … and were not able to distinguish genetically modified mice from normal mice of the control group — almost all of them, especially bald, overgrown hair.


Researchers struck not only by the fact of restoration of hair, but also extremely long-term effect of the drug — was administered for five days, one injection per day, and the action of the drug affected even after four months. Given that the mice do not live more than two years, according to human standards it for a decade.

Subsequent experiments confirmed the original result. When astressin-B began to give young genetically altered mice that have not yet had time to grow bald, a drug to prevent hair loss.

It has long been known that stress causes not only graying, but also baldness. But so far, none of the wonder-means based on the sometimes anti-stress drugs, did not lead to the full restoration of hair and did not make such a long exposure. Until now, only the drug was tested in mice, and whether it will act in the same manner on the people still unknown. But hopes for that. First, the bald mutant mice were trying to cure for baldness "human" drug. He acted in rodents as well as the effect on people — with modest results. Secondly, there is a stress hormone, and human skin, so it can be assumed that the mechanism of alopecia in humans and in mice is the same.

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